Nanomedicine and Lipid Nanoparticles in Atherosclerotic Plaque Regression and Vascular Remodeling

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Medically Reviewed by Valley Clinic Editorial & Biotechnology Advisory Board
Compliant with clinical cGMP, NIH guidelines, and peer-reviewed pharmacology literature.

Atherosclerosis is fundamentally an inflammatory lipid-driven disease of the arterial intima. Next-generation biotechnology approaches focus on actively stabilizing vulnerable fibroatheromas and clearing lipid necrotic cores.

Targeted Macrophage Nanomedicine

Biocompatible nanoparticles engineered with scavenger receptor ligands selectively accumulate in inflamed arterial plaques. Upon endocytosis by lipid-laden foam cells, these nanocarriers deliver anti-inflammatory siRNA or LXR agonists, stimulating reverse cholesterol transport and reducing necrotic core volume.

Small Interfering RNA (siRNA) Gene Silencing: Inclisiran and Beyond

Targeted siRNA conjugates (GalNAc-siRNA) specifically silence hepatic PCSK9 mRNA translation, maintaining low LDL-C receptor recycling cycles and providing sustained 50%+ LDL-C reductions with twice-yearly subcutaneous administration.